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METHYLTESTOSTERONE

Methyltestosterone is one of the oldest known and most popular steroids. Although it is internationally controversial whether it is a prohormone or an oral ANABOLIC steroid, since it is quasi only in the body converted to a steroid due to the composition. Nonetheless, it is an extremely potent remedy for various purposes. All you need to know about this is here:

GENERAL INFORMATION ON METHYLTESTOSTERONE

Methyltestosterone is one of the oldest oral anabolic steroids at all. After about 1930 the structure of the body testosterone was able to be deciphered, one of the next steps was to make the active substance testosterone available orally. Since pure testosterone is almost completely destroyed in oral administration during the so-called first pass by the liver, certain modifications have been necessary for oral availability for the testosterone molecule. Thus the methyltestosterone arose. Since methyltestosterone is a very old active substance with many side effects, it is all the more surprising that it has been so popular both officially and unofficially.

EFFECTS OF METHYLTESTOSTERONE

Methyltestosterone is popular because of a certain property in many power sportsmen and bodybuilders. Methyltestosterone significantly increases both strength and aggression in the short term after administration. For this reason, methyltestosterone is often taken before a particularly intensive training unit or a competition.

DOSAGE OF METHYLTESTOSTERONE

Typical observed dosages are in the range of 25 to 50 mg of methyltestosterone about 60 minutes before the desired effect onset. The sublingual use of methyltestosterone is also popular, since this accelerates the uptake of the active substance. Those who take methyltestosterone in spite of all the contradictory reasons for the muscle build-up use in practice mostly dosages in the range of 50 - 100 mg methyltestosterone per day.

SIDE EFFECTS OF METHYLTESTOSTERONE

Although methyltestosterone can not be as efficiently converted into estrogen by the aromatase enzyme as testosterone, methyltestosterone has strong pronounced estrogen-related side effects, which are manifested by strong water retention, increased blood pressure and an increased risk of gynecomastia. This is due to the fact that methyltestosterone is converted by the aromatase enzyme into 17-methylestradiol, which, like methyltestosterone, can only be degraded poorly by the liver and is therefore significantly longer active in the body than estrogen. In addition to the estrogen-induced side effects, methyltestosterone also has pronounced androgenic side effects which are manifested in, among other things, oily skin, acne, increased body hair, hair loss and an increased risk of prostate enlargement. Here too, the 17-alpha alkylation plays a reinforcing role. The reductase enzyme converts methyltestosterone into 17-alpha-methyldihydro-testosterone instead of dihydrotestosterone, which, like 17-methylestradiol, can only be degraded poorly by the liver.

Structurally, the anabolic androgenic steroid methyltestosterone is a testosterone molecule that has been made available orally by attaching a methyl group at position 17. Because of this 17-alpha alkylation, methyltestosterone, like all other 17-alpha-alkylated steroids, is potentially liver-damaging, which is why in most cases its use is limited to a maximum of 6 to 8 weeks. Methyltestosterone is mediocre androgenic and anabolic, with the androgenic effect weaker than that of testosterone.

Since the anabolic androgenic steroid methyltestosterone, as already described, has a very strong potential for side effects and, due to its short half-life and only moderate anabolic effect, should be taken several times daily in relatively high quantities, it does not represent a particularly good choice of the risk / benefit ratio. The muscle mass built-up with methyltestosterone is heavily influenced by water retention and is lost to a large extent after dicontinuation, since on the one hand the stored water is excreted and, on the other hand, the body testosterone production by methyltestosterone is very efficiently suppressed. Of course, one could counteract the side effects with an antiestrogen such as tamoxifen or better with an aromatase inhibitor such as Arimidex, Aromasin or Femara in combination with a reductase inhibitor such as finasteride. The fact that the costs of these concomitant products, which only fight the side effects, would far exceed the cost of the actual active substance methyltestosterone, and that fewer-side-effects and more effective anabolic androgenic steroids can be obtained for less money, makes the whole use very unattractive.

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